Светлый фон

190 V. I. Sayin et al., “Antioxidants accelerate lung cancer progression in mice.” Science Translational Medicine 6, no. 221 (January 2014): 221ra15.

Science Translational Medicine

191 W. Yang et al., “A mitochondrial superoxide signal triggers increased longevity in Caenorhabditis elegans.” PLoS Biology 8, no. 12 (January 2010): e1000556.

PLoS Biology

192 M. H. Eskelinen et al., “Midlife coffee and tea drinking and the risk of late-life dementia: a population-based CAIDE study.” Journal of Alzheimer’s Disease 16, no. 1 (January 2009): 85–91.

Journal of Alzheimer’s Disease

193 G. W. Ross et al., “Association of coffee and caffeine intake with the risk of Parkinson disease.” JAMA 283, no. 20 (May 2000): 2674–79.

JAMA

194 E. Salazar-Martinez et al., “Coffee consumption and risk for type 2 diabetes mellitus.” Annals of Internal Medicine 140, no. 1 (January 2004): 1–8.

Annals of Internal Medicine

195 M. S. Butt et al., “Coffee and its consumption: benefits and risks.” Critical Reviews in Food Science and Nutrition 51, no. 4 (April 2011): 363–73.

Critical Reviews in Food Science and Nutrition

196 U. Boettler et al., “Coffee constituents as modulators of Nrf2 nuclear translocation and ARE (EpRE)-dependent gene expression.” Journal of Nutritional Biochemistry 22, no. 5 (May 2011): 426–40.

Journal of Nutritional Biochemistry

197 K. Trinh et al., “Induction of the phase II detoxification pathway suppresses neuron loss in Drosophilamodels of Parkinson’s disease.” Journal of Neuroscience: The Official Journal of the Society for Neuroscience 28, no. 2 (January 2008): 465–72.

Journal of Neuroscience: The Official Journal of the Society for Neuroscience

198 M. J. Steinbaugh et al., “Activation of genes involved in xenobiotic metabolism is a shared signature of mouse models with extended lifespan.” American Journal of Physiology Endocrinology and Metabolism 303, no. 4 (August 2012): E488–95.

American Journal of Physiology Endocrinology and Metabolism

199 S. Ayyadevara et al., “Lifespan and stress resistance of Caenorhabditis elegans are increased by expression of glutathione transferases capable of metabolizing the lipid peroxidation product 4-hydroxynonenal.” Aging Cell 4, no. 5 (October 2005): 257–71.

Caenorhabditis elegans